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Lipopolysaccharides Up-regulate Kir6.1/SUR2B Channel Expression and Enhance Vascular K(ATP) Channel Activity via NF-κB-dependent Signaling
Sepsis is a severe medical condition causing a large number of deaths worldwide. Recent studies indicate that the septic susceptibility is attributable to the vascular ATP-sensitive K(+) (K(ATP)) channel. However, the mechanisms underlying the channel modulation in sepsis are still unclear. Here we...
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| Autors principals: | , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Society for Biochemistry and Molecular Biology
2010
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2823456/ https://ncbi.nlm.nih.gov/pubmed/19959479 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M109.058313 |
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