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Calmodulin kinase II is required for angiotensin II-mediated vascular smooth muscle hypertrophy

Despite our understanding that medial smooth muscle hypertrophy is a central feature of vascular remodeling, the molecular pathways underlying this pathology are still not well understood. Work over the past decade has illustrated a potential role for the multifunctional calmodulin-dependent kinase...

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Bibliografiska uppgifter
Huvudupphovsmän: Li, Hui, Li, Weiwei, Gupta, Arun K., Mohler, Peter J., Anderson, Mark E., Grumbach, Isabella M.
Materialtyp: Artigo
Språk:Inglês
Publicerad: American Physiological Society 2010
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Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC2822565/
https://ncbi.nlm.nih.gov/pubmed/20023119
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.01014.2009
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