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Nitric oxide originating from NOS1 controls oxygen utilization and electrolyte transport efficiency in the diabetic kidney
Nitric oxide (NO) is a potent regulator of both vascular tone and cellular oxygen consumption (Qo(2)). Diabetic kidneys have reduced NO availability and increased Qo(2). However, the exact nitric oxide synthase (NOS) isoform regulating Qo(2), hemodynamics, and excretory function in the diabetic kidn...
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| 主要な著者: | , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Physiological Society
2009
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2822516/ https://ncbi.nlm.nih.gov/pubmed/19923416 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00229.2009 |
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