Nitric oxide originating from NOS1 controls oxygen utilization and electrolyte transport efficiency in the diabetic kidney

Nitric oxide (NO) is a potent regulator of both vascular tone and cellular oxygen consumption (Qo(2)). Diabetic kidneys have reduced NO availability and increased Qo(2). However, the exact nitric oxide synthase (NOS) isoform regulating Qo(2), hemodynamics, and excretory function in the diabetic kidn...

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Hlavní autoři: Palm, Fredrik, Fasching, Angelica, Hansell, Peter, Källskog, Örjan
Médium: Artigo
Jazyk:Inglês
Vydáno: American Physiological Society 2009
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Articles
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC2822516/
https://ncbi.nlm.nih.gov/pubmed/19923416
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00229.2009
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