Functional characterization of bovine TIRAP and MyD88 in mediating bacterial lipopolysaccharide-induced endothelial NF-κB activation and apoptosis

Documenti analoghi

• Lipopolysaccharide-induced NF-κB nuclear translocation is primarily dependent on MyD88, but TNFα expression requires TRIF and MyD88
  di: Sakai, Jiro, et al.
  Pubblicazione: (2017)
• PYK2 interacts with MyD88 and regulates MyD88-mediated NF-κB activation in macrophages
  di: Xi, Cai-Xia, et al.
  Pubblicazione: (2010)
• Biochanin A Reduces Inflammatory Injury and Neuronal Apoptosis following Subarachnoid Hemorrhage via Suppression of the TLRs/TIRAP/MyD88/NF-κB Pathway
  di: Wu, Ling-yun, et al.
  Pubblicazione: (2018)
• NF-κB mediates lipopolysaccharide-induced alternative pre-mRNA splicing of MyD88 in mouse macrophages
  di: Lee, Frank Fang-Yao, et al.
  Pubblicazione: (2020)
• Penta-O-galloyl-β-D-glucose ameliorates inflammation by inhibiting MyD88/NF-κB and MyD88/MAPK signalling pathways
  di: Jang, Se-Eun, et al.
  Pubblicazione: (2013)