Axonal Transport Rates In Vivo Are Unaffected by Tau Deletion or Overexpression in Mice

Elevated tau expression has been proposed as a possible basis for impaired axonal transport in Alzheimer's disease. To address this hypothesis, we analyzed the movement of pulse radiolabeled proteins in vivo along retinal ganglion cell (RGC) axons of mice that lack tau or overexpress human tau...

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Main Authors: Yuan, Aidong, Kumar, Asok, Peterhoff, Corrinne, Duff, Karen, Nixon, Ralph A.
Formáid: Artigo
Teanga:Inglês
Foilsithe: Society for Neuroscience 2008
Ábhair:
Brief Communications
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC2814454/
https://ncbi.nlm.nih.gov/pubmed/18272688
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.5242-07.2008
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