Rosiglitazone via upregulation of Akt/eNOS pathways attenuates dysfunction of endothelial progenitor cells, induced by advanced glycation end products

BACKGROUND AND PURPOSE: Advanced glycation end products (AGEs) and endothelial progenitor cells (EPCs) play key roles in pathogenesis of diabetes-related vascular complications. AGEs can induce dysfunction in EPCs. The peroxisome proliferator-activated receptor-gamma (PPARγ) agonists are widely used...

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Bibliografiske detaljer
Main Authors: Liang, Chun, Ren, Yusheng, Tan, Hongbin, He, Zhiqing, Jiang, Qijun, Wu, Jianxiang, Zhen, Yi, Fan, Min, Wu, Zonggui
Format: Artigo
Sprog:Inglês
Udgivet: Blackwell Publishing Ltd 2009
Fag:
Research Papers
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2807648/
https://ncbi.nlm.nih.gov/pubmed/19917066
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1476-5381.2009.00450.x
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