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Helicobacter pylori Lipopolysaccharides Upregulate Toll-Like Receptor 4 Expression and Proliferation of Gastric Epithelial Cells via the MEK1/2-ERK1/2 Mitogen-Activated Protein Kinase Pathway

Helicobacter pylori is recognized as an etiological agent of gastroduodenal diseases. H. pylori produces various toxic substances, including lipopolysaccharide (LPS). However, H. pylori LPS exhibits extremely weakly endotoxic activity compared to the typical LPS, such as that produced by Escherichia...

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Detalhes bibliográficos
Main Authors: Yokota, Shin-ichi, Okabayashi, Tamaki, Rehli, Michael, Fujii, Nobuhiro, Amano, Ken-ichi
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Microbiology (ASM) 2010
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2798195/
https://ncbi.nlm.nih.gov/pubmed/19858308
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/IAI.00903-09
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