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O-GlcNAc signaling attenuates ER stress-induced cardiomyocyte death
We previously demonstrated that the O-linked β-N-acetylglucosamine (O-GlcNAc) posttranslational modification confers cardioprotection at least partially through mitochondrial-dependent mechanisms, but it remained unclear if O-GlcNAc signaling interfered with other mechanisms of cell death. Because i...
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| Main Authors: | , , , |
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| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
American Physiological Society
2009
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2781370/ https://ncbi.nlm.nih.gov/pubmed/19734355 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00553.2009 |
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