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O-GlcNAc signaling attenuates ER stress-induced cardiomyocyte death

We previously demonstrated that the O-linked β-N-acetylglucosamine (O-GlcNAc) posttranslational modification confers cardioprotection at least partially through mitochondrial-dependent mechanisms, but it remained unclear if O-GlcNAc signaling interfered with other mechanisms of cell death. Because i...

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Hlavní autoři: Ngoh, Gladys A., Hamid, Tariq, Prabhu, Sumanth D., Jones, Steven P.
Médium: Artigo
Jazyk:Inglês
Vydáno: American Physiological Society 2009
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC2781370/
https://ncbi.nlm.nih.gov/pubmed/19734355
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00553.2009
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