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Endogenous lipid- and peptide-derived anti-inflammatory pathways generated with glucocorticoid and aspirin treatment activate the lipoxin A(4) receptor

Aspirin (ASA) and dexamethasone (DEX) are widely used anti-inflammatory agents yet their mechanism(s) for blocking polymorphonuclear neutrophil (PMN) accumulation at sites of inflammation remains unclear. Here, we report that inhibition of PMN infiltration by ASA and DEX is a property shared by aspi...

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Bibliografski detalji
Glavni autori: Perretti, Mauro, Chiang, Nan, La, Mylinh, Fierro, Iolanda M., Marullo, Stefano, Getting, Stephen J, Solito, Egle, Serhan, Charles N.
Format: Artigo
Jezik:Inglês
Izdano: 2002
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC2777269/
https://ncbi.nlm.nih.gov/pubmed/12368905
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nm786
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