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System-Level Control to Optimize Glucagon Counterregulation by Switch-Off of α-Cell Suppressing Signals in β-Cell Deficiency

BACKGROUND: Glucagon counterregulation (GCR) is a key protection against hypoglycemia that is compromised in diabetes. In β-cell-deficient rats, GCR pulsatility can be amplified if insulin (INS) or somatostatin (SS) are infused in the pancreatic artery and then switched off during hypoglycemia. The...

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Autors principals: Farhy, Leon S., McCall, Anthony L.
Format: Artigo
Idioma:Inglês
Publicat: Diabetes Technology Society 2009
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC2769841/
https://ncbi.nlm.nih.gov/pubmed/20046648
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