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Zinc Inhibits Amyloid β Production from Alzheimer's Amyloid Precursor Protein in SH-SY5Y Cells
Zinc released from excited glutamatergic neurons accelerates amyloid β (Aβ) aggregation, underscoring the therapeutic potential of zinc chelation for the treatment of Alzheimer's disease (AD). Zinc can also alter Aβ concentration by affecting its degradation. In order to elucidate the possible...
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| Autors principals: | , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
The Korean Physiological Society and The Korean Society of Pharmacology
2009
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2766728/ https://ncbi.nlm.nih.gov/pubmed/19885037 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4196/kjpp.2009.13.3.195 |
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