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Disruption of glutamate receptors at Shank-postsynaptic platform in Alzheimer's disease
Synaptic loss underlies the memory deficit of Alzheimer's disease (AD). The molecular mechanism is elusive; however, excitatory synapses organized by the postsynaptic density (PSD) have been used as targets for AD treatment. To identify pathological entities at the synapse in AD, synaptic prote...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
2009
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2745956/ https://ncbi.nlm.nih.gov/pubmed/19635471 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.brainres.2009.07.056 |
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