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Endothelial nitric oxide synthase decreases β-adrenergic responsiveness via inhibition of the L-type Ca(2+) current
Signaling via endothelial nitric oxide synthase (NOS3) limits the heart’s response to β-adrenergic (β-AR) stimulation, which may be protective against arrhythmias. However, mechanistic data are limited. Therefore, we performed simultaneous measurements of action potential (AP, using patch clamp), Ca...
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| Main Authors: | , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2008
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2744450/ https://ncbi.nlm.nih.gov/pubmed/18203845 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.01249.2007 |
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