Estradiol-Induced Regression in T47D:A18/PKCα Tumors Requires the Estrogen Receptor And Interaction with the Extracellular Matrix

Several breast cancer tumor models respond to estradiol (E2) by undergoing apoptosis, a phenomenon known to occur in clinical breast cancer. Prior to the application of tamoxifen as an endocrine therapy, high dose E2 or diethystilbesterol (DES) treatment was successfully utilized, albeit with unfavo...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Zhang, Yiyun, Zhao, Huiping, Asztalos, Szilard, Chisamore, Michael, Sitabkhan, Yasmin, Tonetti, Debra A.
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: 2009
Gaiak:
Article
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC2743931/
https://ncbi.nlm.nih.gov/pubmed/19372579
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/1541-7786.MCR-08-0415
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