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Alternative splicing of RyR1 alters the efficacy of skeletal EC coupling
Alternative splicing of ASI residues (Ala(3481)-Gln(3485)) in the skeletal muscle ryanodine receptor (RyR1) is developmentally regulated: the residues are present in adult ASI(+)RyR1, but absent in the juvenile ASI(-)RyR1 which is over-expressed in adult myotonic dystrophy type 1 (DM1). Although thi...
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| Hoofdauteurs: | , , , , , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
2009
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2743929/ https://ncbi.nlm.nih.gov/pubmed/19131108 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ceca.2008.11.005 |
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