Hemizygous Deficiency of Kruppel-like Factor 2 Augments Experimental Atherosclerosis

Kruppel-like factor 2 (KLF2) is a central regulator of endothelial and monocyte/macrophage gene expression and function in vitro. While the composite effects of KLF2 in these two cell types predict that it likely inhibits vascular inflammation, the role of KLF2 in this process in vivo is uncharacter...

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Detaylı Bibliyografya
Asıl Yazarlar: Atkins, G. Brandon, Wang, Yunmei, Mahabeleshwar, Ganapati H., Shi, Hong, Gao, Huiyun, Kawanami, Daiji, Natesan, Viswanath, Lin, Zhiyong, Simon, Daniel I., Jain, Mukesh K.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2008
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2743294/
https://ncbi.nlm.nih.gov/pubmed/18757824
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCRESAHA.108.184663
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