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Distinction of the memory B cell response to cognate antigen versus bystander inflammatory signals
The hypothesis that bystander inflammatory signals promote memory B cell (B(MEM)) self-renewal and differentiation in an antigen-independent manner is critically evaluated herein. To comprehensively address this hypothesis, a detailed analysis is presented examining the response profiles of B-2 line...
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Hoofdauteurs: | , , , , , , |
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Formaat: | Artigo |
Taal: | Inglês |
Gepubliceerd in: |
The Rockefeller University Press
2009
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Onderwerpen: | |
Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2737154/ https://ncbi.nlm.nih.gov/pubmed/19703988 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1084/jem.20090667 |
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