Loss of positive allosteric interactions between neuronal nitric oxide synthase and phosphofructokinase contributes to defects in glycolysis and increased fatigability in muscular dystrophy

Duchenne muscular dystrophy (DMD) involves a complex pathophysiology that is not easily explained by the loss of the protein dystrophin, the primary defect in DMD. Instead, many features of the pathology are attributable to the secondary loss of neuronal nitric oxide synthase (nNOS) from dystrophin-...

Full beskrivning

Sparad:
Bibliografiska uppgifter
Huvudupphovsmän: Wehling-Henricks, Michelle, Oltmann, Meredith, Rinaldi, Chiara, Myung, Kyu H., Tidball, James G.
Materialtyp: Artigo
Språk:Inglês
Publicerad: Oxford University Press 2009
Ämnen:
Articles
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC2729666/
https://ncbi.nlm.nih.gov/pubmed/19542095
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/hmg/ddp288
Taggar: Lägg till en tagg
Inga taggar, Lägg till första taggen!

Liknande verk