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A nitric oxide synthase transgene ameliorates muscular dystrophy in mdx mice

Dystrophin-deficient muscles experience large reductions in expression of nitric oxide synthase (NOS), which suggests that NO deficiency may influence the dystrophic pathology. Because NO can function as an antiinflammatory and cytoprotective molecule, we propose that the loss of NOS from dystrophic...

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書目詳細資料
Main Authors: Wehling, Michelle, Spencer, Melissa J., Tidball, James G.
格式: Artigo
語言:Inglês
出版: The Rockefeller University Press 2001
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在線閱讀:https://ncbi.nlm.nih.gov/pmc/articles/PMC2150800/
https://ncbi.nlm.nih.gov/pubmed/11581289
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1083/jcb.200105110
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