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A nitric oxide synthase transgene ameliorates muscular dystrophy in mdx mice

Dystrophin-deficient muscles experience large reductions in expression of nitric oxide synthase (NOS), which suggests that NO deficiency may influence the dystrophic pathology. Because NO can function as an antiinflammatory and cytoprotective molecule, we propose that the loss of NOS from dystrophic...

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Bibliografiske detaljer
Main Authors: Wehling, Michelle, Spencer, Melissa J., Tidball, James G.
Format: Artigo
Sprog:Inglês
Udgivet: The Rockefeller University Press 2001
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2150800/
https://ncbi.nlm.nih.gov/pubmed/11581289
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1083/jcb.200105110
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