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GRP78 Upregulation by Atheroprone Shear Stress via p38-, α2β1-dependent Mechanism in Endothelial Cells
OBJECTIVE: The initiation of atherosclerosis is in part dependent on the hemodynamic shear stress environment promoting a pro-inflammatory phenotype of the endothelium. Previous studies demonstrated increased expression of ER stress protein and unfolded protein response (UPR) regulator, GRP78, withi...
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| Main Authors: | , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2008
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2723835/ https://ncbi.nlm.nih.gov/pubmed/18556570 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/ATVBAHA.108.167999 |
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