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Investigation of the Mechanism by Which Herpes Simplex Virus Type 1 LAT Sequences Modulate Preferential Establishment of Latent Infection in Mouse Trigeminal Ganglia

We previously demonstrated that herpes simplex virus type 1 (HSV-1) preferentially establishes latent infection in monoclonal antibody (MAb) A5-positive ganglionic neurons and that a 2.8-kb portion of the HSV-1 genome, corresponding to the 5′ end of the LAT (latency-associated transcript) coding reg...

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Hlavní autoři: Imai, Yumi, Apakupakul, Kathleen, Krause, Philip R., Halford, William P., Margolis, Todd P.
Médium: Artigo
Jazyk:Inglês
Vydáno: American Society for Microbiology (ASM) 2009
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On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC2715745/
https://ncbi.nlm.nih.gov/pubmed/19493993
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/JVI.00043-09
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