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Tumour necrosis factor α activates nuclear factor κB signalling to reduce N-type voltage-gated Ca(2+) current in postganglionic sympathetic neurons
Inflammation has profound effects on the innervation of affected tissues, including altered neuronal excitability and neurotransmitter release. As Ca(2+) influx through voltage-gated Ca(2+) channels (VGCCs) is a critical determinant of excitation–secretion coupling in nerve terminals, the aim of thi...
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| Hauptverfasser: | , , , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
Blackwell Science Inc
2009
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2714026/ https://ncbi.nlm.nih.gov/pubmed/19403618 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2009.172312 |
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