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AIP-1 ameliorates β-amyloid peptide toxicity in a Caenorhabditis elegans Alzheimer's disease model
Multiple neurodegenerative diseases are causally linked to aggregation-prone proteins. Cellular mechanisms involving protein turnover may be key defense mechanisms against aggregating protein disorders. We have used a transgenic Caenorhabditis elegans Alzheimer's disease model to identify cellu...
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主要な著者: | , , , |
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フォーマット: | Artigo |
言語: | Inglês |
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Oxford University Press
2009
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オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2706681/ https://ncbi.nlm.nih.gov/pubmed/19414486 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/hmg/ddp209 |
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