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AIP-1 ameliorates β-amyloid peptide toxicity in a Caenorhabditis elegans Alzheimer's disease model

Multiple neurodegenerative diseases are causally linked to aggregation-prone proteins. Cellular mechanisms involving protein turnover may be key defense mechanisms against aggregating protein disorders. We have used a transgenic Caenorhabditis elegans Alzheimer's disease model to identify cellu...

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Autors principals: Hassan, Wail M., Merin, David A., Fonte, Virginia, Link, Christopher D.
Format: Artigo
Idioma:Inglês
Publicat: Oxford University Press 2009
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC2706681/
https://ncbi.nlm.nih.gov/pubmed/19414486
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/hmg/ddp209
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