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The basal flux of Akt in the mitochondria is mediated by heat shock protein-90
Akt is a known client protein of heat shock protein-90 (HSP90). We have found that HSP90 is responsible for Akt accumulation in the mitochondria in unstimulated cells. Treatment of SH-SY5Y neuroblastoma cells and HEK293 cells with the HSP90 inhibitors novobiocin and geldanamycin caused substantial d...
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| Autors principals: | , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2009
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2696161/ https://ncbi.nlm.nih.gov/pubmed/19187436 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1471-4159.2009.05878.x |
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