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Nuclear export of Smad2 and Smad3 by RanBP3 facilitates termination of TGF-β signaling

Smad2 and Smad3 (Smad2/3) are key intracellular signal transducers for TGF-β signaling and their transcriptional activities are controlled through reversible phosphorylation and nucleocytoplasmic shuttling. However, the precise mechanism underlying nuclear export of Smad2/3 remains elusive. Here we...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Dai, Fangyan, Lin, Xia, Chang, Chenbei, Feng, Xin-Hua
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2009
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2676691/
https://ncbi.nlm.nih.gov/pubmed/19289081
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.devcel.2009.01.022
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