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Erythromycin resistance by L4/L22 mutations and resistance masking by drug efflux pump deficiency
We characterized the effects of classical erythromycin resistance mutations in ribosomal proteins L4 and L22 of the large ribosomal subunit on the kinetics of erythromycin binding. Our data are consistent with a mechanism in which the macrolide erythromycin enters and exits the ribosome through the...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group
2009
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2666033/ https://ncbi.nlm.nih.gov/pubmed/19197244 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/emboj.2009.17 |
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