טוען...
Rif1 and Rif2 inhibit localization of Tel1 to DNA ends
Chromosome ends, known as telomeres, have to be distinguished from DNA double-strand breaks (DSBs) that activate the DNA damage checkpoint. In budding yeast, the ATM homolog Tel1 associates preferentially with short telomeres and promotes telomere addition. Here we show that the telomeric proteins R...
שמור ב:
| Main Authors: | , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2009
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2662776/ https://ncbi.nlm.nih.gov/pubmed/19217405 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molcel.2008.12.027 |
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