Diverse Functional Consequences of Mutations in the Na(+)/K(+)-ATPase α(2)-Subunit Causing Familial Hemiplegic Migraine Type 2

Similar Items

• E(2)P State Stabilization by the N-terminal Tail of the H,K-ATPase β-Subunit Is Critical for Efficient Proton Pumping under in Vivo Conditions
  od: Dürr, Katharina L., i dr.
  Izdano: (2009)
• Hyperpolarization-activated inward leakage currents caused by deletion or mutation of carboxy-terminal tyrosines of the Na(+)/K(+)-ATPase α subunit
  od: Meier, Susan, i dr.
  Izdano: (2010)
• Control of Gastric H,K-ATPase Activity by Cations, Voltage and Intracellular pH Analyzed by Voltage Clamp Fluorometry in Xenopus Oocytes
  od: Dürr, Katharina L., i dr.
  Izdano: (2012)
• Measuring Cation Transport by Na,K- and H,K-ATPase in Xenopus Oocytes by Atomic Absorption Spectrophotometry: An Alternative to Radioisotope Assays
  od: Dürr, Katharina L., i dr.
  Izdano: (2013)
• Inhibition of Phosphorylation of Na(+),K(+)-ATPase by Mutations Causing Familial Hemiplegic Migraine
  od: Schack, Vivien Rodacker, i dr.
  Izdano: (2012)