IL-32–dependent effects of IL-1β on endothelial cell functions

Increasing evidence demonstrates that interleukin (IL)–32 is a pro-inflammatory cytokine, inducing IL-1α, IL-1β, IL-6, tumor necrosis factor (TNF)–α, and chemokines via nuclear factor (NF)–κB, p38 mitogen-activated protein kinase (MAPK), and activating protein (AP)-1 activation. Here we report that...

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Detaylı Bibliyografya
Asıl Yazarlar: Nold-Petry, Claudia A., Nold, Marcel F., Zepp, Jarod A., Kim, Soo-Hyun, Voelkel, Norbert F., Dinarello, Charles A.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: National Academy of Sciences 2009
Konular:
Biological Sciences
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2656174/
https://ncbi.nlm.nih.gov/pubmed/19228941
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0813334106
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