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An Expanded Biological Repertoire for Ins(3,4,5,6)P(4) through its Modulation of ClC-3 Function
Ins(3,4,5,6)P(4) inhibits plasma membrane Cl(−) flux in secretory epithelia [1]. However, in most other mammalian cells, receptor-dependent elevation of Ins(3,4,5,6)P(4) levels is an “orphan” response that lacks biological significance [2]. We set out to identify Cl(−) channel(s) and/or transporter(...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2008
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2631652/ https://ncbi.nlm.nih.gov/pubmed/18951024 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cub.2008.08.073 |
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