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Coupling of endoplasmic reticulum stress to CDDO-Me-induced upregulation of death receptor 5 via a CHOP-dependent mechanism involving JNK activation

The synthetic triterpenoid methyl 2-cyano-3,12-dioxoolean-1,9-dien-28-oate (CDDO-Me) is in Phase I clinical trials as a novel cancer therapeutic agent. We previously demonstrated that CDDO-Me induces c-Jun N-terminal kinase (JNK)-dependent death receptor 5 (DR5) expression and augments death recepto...

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Autori principali: Zou, Wei, Yue, Ping, Khuri, Fadlo R., Sun, Shi-Yong
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2008
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC2597446/
https://ncbi.nlm.nih.gov/pubmed/18794136
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/0008-5472.CAN-08-1318
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