Kinin B(2 )receptor regulates chemokines CCL2 and CCL5 expression and modulates leukocyte recruitment and pathology in experimental autoimmune encephalomyelitis (EAE) in mice

BACKGROUND: Kinins are important mediators of inflammation and act through stimulation of two receptor subtypes, B(1 )and B(2). Leukocyte infiltration contributes to the pathogenesis of autoimmune inflammation in the central nervous system (CNS), occurring not only in multiple sclerosis (MS) but als...

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保存先:
書誌詳細
主要な著者: Dos Santos, Adriana C, Roffê, Ester, Arantes, Rosa ME, Juliano, Luiz, Pesquero, Jorge L, Pesquero, João B, Bader, Michael, Teixeira, Mauro M, Carvalho-Tavares, Juliana
フォーマット: Artigo
言語:Inglês
出版事項: BioMed Central 2008
主題:
Research
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC2596102/
https://ncbi.nlm.nih.gov/pubmed/18986535
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1742-2094-5-49
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