Kinin B(2 )receptor regulates chemokines CCL2 and CCL5 expression and modulates leukocyte recruitment and pathology in experimental autoimmune encephalomyelitis (EAE) in mice

BACKGROUND: Kinins are important mediators of inflammation and act through stimulation of two receptor subtypes, B(1 )and B(2). Leukocyte infiltration contributes to the pathogenesis of autoimmune inflammation in the central nervous system (CNS), occurring not only in multiple sclerosis (MS) but als...

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Main Authors:	Dos Santos, Adriana C, Roffê, Ester, Arantes, Rosa ME, Juliano, Luiz, Pesquero, Jorge L, Pesquero, João B, Bader, Michael, Teixeira, Mauro M, Carvalho-Tavares, Juliana
Format:	Artigo
Sprog:	Inglês
Udgivet:	BioMed Central 2008
Fag:	Research
Online adgang:	https://ncbi.nlm.nih.gov/pmc/articles/PMC2596102/ https://ncbi.nlm.nih.gov/pubmed/18986535 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1742-2094-5-49
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Kinin B(2 )receptor regulates chemokines CCL2 and CCL5 expression and modulates leukocyte recruitment and pathology in experimental autoimmune encephalomyelitis (EAE) in mice

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