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p53 induces distinct epigenetic states at its direct target promoters
BACKGROUND: The tumor suppressor protein p53 is a transcription factor that is mutated in many cancers. Regulation of gene expression by binding of wild-type p53 to its target sites is accompanied by changes in epigenetic marks like histone acetylation. We studied DNA binding and epigenetic changes...
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| Main Authors: | , , , |
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| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
BioMed Central
2008
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2585595/ https://ncbi.nlm.nih.gov/pubmed/18922183 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1471-2164-9-486 |
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