Nitric Oxide Promotes Caspase-Independent Hepatic Stellate Cell Apoptosis Through the Generation of Reactive Oxygen Species

Hepatic stellate cells (HSCs) contribute to portal hypertension through multiple mechanisms that include collagen deposition, vasoconstriction, and regulation of sinusoidal structure. Under normal physiologic conditions, endothelial nitric oxide (NO) synthase–derived NO exerts paracrine effects on H...

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Detalhes bibliográficos
Main Authors: Langer, Daniel A., Das, Amitava, Semela, David, Kang-Decker, Ningling, Hendrickson, Helen, Bronk, Steven F., Katusic, Zvonimir S., Gores, Gregory J., Shah, Vijay H.
Formato: Artigo
Idioma:Inglês
Publicado em: 2008
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2562502/
https://ncbi.nlm.nih.gov/pubmed/18459124
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/hep.22285
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