Activation of mutated simian virus 40 enhancers by amplification of wild-type enhancer elements.
We show that duplication of any one of three separate simian virus 40 enhancer elements, A, B, or C, can compensate for loss of function in the remaining two. Simian virus 40 revertants containing point mutations within the A and C (dpm16) or B and C (dpm26) enhancer elements contain tandem duplicat...
保存先:
| 出版年: | J Virol |
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| 主要な著者: | , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Microbiology (ASM)
1987
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC255952/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2822961/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.61.11.3536-3542.1987 |
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