Activation of mutated simian virus 40 enhancers by amplification of wild-type enhancer elements.

We show that duplication of any one of three separate simian virus 40 enhancer elements, A, B, or C, can compensate for loss of function in the remaining two. Simian virus 40 revertants containing point mutations within the A and C (dpm16) or B and C (dpm26) enhancer elements contain tandem duplicat...

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Veröffentlicht in:	J Virol
Hauptverfasser:	Clarke, J, Herr, W
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	American Society for Microbiology (ASM) 1987
Schlagworte:	Research Article
Online-Zugang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC255952/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2822961/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.61.11.3536-3542.1987
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