High oxygen prevents fetal lethality due to lack of catecholamines

The catecholamine norepinephrine is required for fetal survival, but its essential function is unknown. When catecholamine-deficient [tyrosine hydroxylase (Th) null] mouse fetuses die at embryonic day (E)13.5–14.5, they resemble wild-type (wt) fetuses exposed to hypoxia. They exhibit bradycardia (28...

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Bibliografiska uppgifter
Huvudupphovsmän: Ream, Margie A., Chandra, Rashmi, Peavey, Mary, Ray, Alisa M., Roffler-Tarlov, Suzanne, Kim, Hyung-Gun, Wetsel, William C., Rockman, Howard A., Chikaraishi, Dona M.
Materialtyp: Artigo
Språk:Inglês
Publicerad: American Physiological Society 2008
Ämnen:
Exercise and Respiratory Physiology
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC2536863/
https://ncbi.nlm.nih.gov/pubmed/18635452
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpregu.00860.2007
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