טוען...
High oxygen prevents fetal lethality due to lack of catecholamines
The catecholamine norepinephrine is required for fetal survival, but its essential function is unknown. When catecholamine-deficient [tyrosine hydroxylase (Th) null] mouse fetuses die at embryonic day (E)13.5–14.5, they resemble wild-type (wt) fetuses exposed to hypoxia. They exhibit bradycardia (28...
שמור ב:
| Main Authors: | , , , , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Physiological Society
2008
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2536863/ https://ncbi.nlm.nih.gov/pubmed/18635452 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpregu.00860.2007 |
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