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Ca(2+)/Calmodulin-dependent Protein Kinase II-dependent Remodeling of Ca(2+) Current in Pressure Overload Heart Failure
Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) activity is increased in heart failure (HF), a syndrome characterized by markedly increased risk of arrhythmia. Activation of CaMKII increases peak L-type Ca(2+) current (I(Ca)) and slows I(Ca) inactivation. Whether these events are linked mecha...
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| Autors principals: | , , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Society for Biochemistry and Molecular Biology
2008
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2533065/ https://ncbi.nlm.nih.gov/pubmed/18622016 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M803043200 |
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