Human Immunodeficiency Virus Infection Alters Tumor Necrosis Factor Alpha Production via Toll-Like Receptor-Dependent Pathways in Alveolar Macrophages and U1 Cells

Human immunodeficiency virus (HIV)-positive persons are predisposed to pulmonary infections, even after receiving effective highly active antiretroviral therapy. The reasons for this are unclear but may involve changes in innate immune function. HIV type 1 infection of macrophages impairs effector f...

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Detalhes bibliográficos
Main Authors: Nicol, Marlynne Q., Mathys, Jean-Marie, Pereira, Albertina, Ollington, Kevin, Ieong, Michael H., Skolnik, Paul R.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Microbiology (ASM) 2008
Assuntos:
Pathogenesis and Immunity
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2519548/
https://ncbi.nlm.nih.gov/pubmed/18524817
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/JVI.00362-08
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