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AGE/RAGE produces endothelial dysfunction in coronary arterioles in Type 2 diabetic mice
We hypothesized that impaired nitric oxide (NO)-dependent dilation (endothelial dysfunction) in Type 2 diabetes results, in part, from elevated production of superoxide (O(2)(•−)) induced by the interaction of advanced glycation end products (AGE)/receptor for AGE (RAGE) and TNF-α signaling. We asse...
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| Autors principals: | , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Physiological Society
2008
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2519223/ https://ncbi.nlm.nih.gov/pubmed/18539754 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00464.2008 |
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