The NF-kappa B binding sites in the human immunodeficiency virus type 1 long terminal repeat are not required for virus infectivity.

Mutations were introduced into the regulatory sequences in the long terminal repeat of an infectious molecular clone of the human immunodeficiency virus. Viruses in which the NF-kappa B binding sites were deleted or ones in which one or two Sp1 binding sites were mutated still replicated efficiently...

תיאור מלא

מידע ביבליוגרפי

הוצא לאור ב:	J Virol
Principais autores:	Leonard, J, Parrott, C, Buckler-White, A J, Turner, W, Ross, E K, Martin, M A, Rabson, A B
פורמט:	Artigo
שפה:	Inglês
יצא לאור:	American Society for Microbiology (ASM) 1989
נושאים:	Research Article
גישה מקוונת:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC251138/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2795721/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.63.11.4919-4924.1989
תגים:	הוספת תג אין תגיות, היה/י הראשונ/ה לתייג את הרשומה!