The NF-kappa B binding sites in the human immunodeficiency virus type 1 long terminal repeat are not required for virus infectivity.

Mutations were introduced into the regulatory sequences in the long terminal repeat of an infectious molecular clone of the human immunodeficiency virus. Viruses in which the NF-kappa B binding sites were deleted or ones in which one or two Sp1 binding sites were mutated still replicated efficiently...

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Detaylı Bibliyografya

Yayımlandı:	J Virol
Asıl Yazarlar:	Leonard, J, Parrott, C, Buckler-White, A J, Turner, W, Ross, E K, Martin, M A, Rabson, A B
Materyal Türü:	Artigo
Dil:	Inglês
Baskı/Yayın Bilgisi:	American Society for Microbiology (ASM) 1989
Konular:	Research Article
Online Erişim:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC251138/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2795721/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.63.11.4919-4924.1989
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