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Chronic diet-induced hyperhomocysteinemia impairs eNOS regulation in mouse mesenteric arteries

Hyperhomocysteinemia (HHcy) impairs endothelium-dependent vasodilation by increasing reactive oxygen species, thereby reducing nitric oxide (NO·) bioavailability. It is unclear whether reduced expression or function of the enzyme that produces NO·, endothelial nitric oxide synthase (eNOS), also cont...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Looft-Wilson, Robin C., Ashley, Blair S., Billig, Janelle E., Wolfert, Madeline R., Ambrecht, Lindsay A., Bearden, Shawn E.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Physiological Society 2008
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2494800/
https://ncbi.nlm.nih.gov/pubmed/18448615
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpregu.00833.2007
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