The ΔF508-CFTR mutation results in increased biofilm formation by Pseudomonas aeruginosa by increasing iron availability

Lignende værker

• Pseudomonas aeruginosa biofilm formation in the cystic fibrosis airway. A short review
  af: Moreau-Marquis, Sophie, et al.
  Udgivet: (2008)
• Tobramycin and FDA-Approved Iron Chelators Eliminate Pseudomonas aeruginosa Biofilms on Cystic Fibrosis Cells
  af: Moreau-Marquis, Sophie, et al.
  Udgivet: (2009)
• Does the ΔF508-CFTR mutation induce a proinflammatory response in human airway epithelial cells?
  af: Hampton, Thomas H., et al.
  Udgivet: (2012)
• Tgf-β1 Inhibits Cftr Biogenesis and Prevents Functional Rescue of ΔF508-Cftr in Primary Differentiated Human Bronchial Epithelial Cells
  af: Snodgrass, Steven M., et al.
  Udgivet: (2013)
• In Vitro Analysis of Tobramycin-Treated Pseudomonas aeruginosa Biofilms on Cystic Fibrosis-Derived Airway Epithelial Cells
  af: Anderson, Gregory G., et al.
  Udgivet: (2008)