Increased protein O-GlcNAc modification inhibits inflammatory and neointimal responses to acute endoluminal arterial injury

Inflammation plays a major role in vascular disease. We have shown that leukocyte infiltration and inflammatory mediator expression contribute to vascular remodeling after endoluminal injury. This study tested whether increasing protein O-linked-N-acetylglucosamine (O-GlcNAc) levels with glucosamine...

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Bibliografische gegevens
Hoofdauteurs: Xing, Dongqi, Feng, Wenguang, Nöt, Laszlo G., Miller, Andrew P., Zhang, Yun, Chen, Yiu-Fai, Majid-Hassan, Erum, Chatham, John C., Oparil, Suzanne
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: American Physiological Society 2008
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Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2494765/
https://ncbi.nlm.nih.gov/pubmed/18469144
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.01259.2007
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