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Rad53 regulates replication fork restart after DNA damage in Saccharomyces cerevisiae
Replication fork stalling at a DNA lesion generates a damage signal that activates the Rad53 kinase, which plays a vital role in survival by stabilizing stalled replication forks. However, evidence that Rad53 directly modulates the activity of replication forks has been lacking, and the nature of fo...
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| Main Authors: | , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Cold Spring Harbor Laboratory Press
2008
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2492737/ https://ncbi.nlm.nih.gov/pubmed/18628397 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/gad.1660408 |
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