Disruption of GM2/GD2 synthase gene resulted in overt expression of 9-O-acetyl GD3 irrespective of Tis211

GM2/GD2 synthase gene knockout mice lack all complex gangliosides, which are abundantly expressed in the nervous systems of vertebrates. In turn, they have increased precursor structures GM3 and GD3, probably replacing the roles of the depleted complex gangliosides. In this study, we found that 9-O-...

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Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Furukawa, Keiko, Aixinjueluo, Wei, Kasama, Takeshi, Ohkawa, Yuki, Yoshihara, Michiko, Ohmi, Yusuke, Tajima, Orie, Suzumura, Akio, Kittaka, Daiji, Furukawa, Koichi
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: Blackwell Publishing Ltd 2008
Gaiak:
Original Articles
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC2408653/
https://ncbi.nlm.nih.gov/pubmed/18194438
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1471-4159.2008.05232.x
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