Disruption of GM2/GD2 synthase gene resulted in overt expression of 9-O-acetyl GD3 irrespective of Tis211

GM2/GD2 synthase gene knockout mice lack all complex gangliosides, which are abundantly expressed in the nervous systems of vertebrates. In turn, they have increased precursor structures GM3 and GD3, probably replacing the roles of the depleted complex gangliosides. In this study, we found that 9-O-...

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Bibliographische Detailangaben
Hauptverfasser: Furukawa, Keiko, Aixinjueluo, Wei, Kasama, Takeshi, Ohkawa, Yuki, Yoshihara, Michiko, Ohmi, Yusuke, Tajima, Orie, Suzumura, Akio, Kittaka, Daiji, Furukawa, Koichi
Format: Artigo
Sprache:Inglês
Veröffentlicht: Blackwell Publishing Ltd 2008
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Original Articles
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2408653/
https://ncbi.nlm.nih.gov/pubmed/18194438
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1471-4159.2008.05232.x
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